Search results for "genetics [Mutation]"

showing 10 items of 3026 documents

Dissecting genome reduction and trait loss in insect endosymbionts

2016

Symbiosis has played a major role in eukaryotic evolution beyond the origin of the eukaryotic cell. Thus, organisms across the tree of life are associated with diverse microbial partners, conferring to the host new adaptive traits that enable it to explore new niches. This is the case for insects thriving on unbalanced diets, which harbor mutualistic intracellular microorganisms, mostly bacteria that supply them with the required nutrients. As a consequence of the lifestyle change, from free-living to host-associated mutualist, a bacterium undergoes many structural and metabolic changes, of which genome shrinkage is the most dramatic. The trend toward genome size reduction in endosymbiotic …

0301 basic medicineGeneticsbiologyGeneral NeuroscienceMicroorganismBacterial genome sizebiology.organism_classificationPhenotypeGenomeGeneral Biochemistry Genetics and Molecular Biology03 medical and health sciences030104 developmental biologyHistory and Philosophy of ScienceSymbiosisGenome sizeGeneBacteriaAnnals of the New York Academy of Sciences
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Evaluation of the RYR1 gene genetic diversity in the Latvian White pig breed

2016

The ryanodine receptor 1 (RYR1) is a calcium ion channel in the sarcoplasmic reticulum of skeletal muscle. Multiple polymorphic loci have been identified in the RYR1 gene in human and animals and some of them are associated with certain phenotypes. However, there are still few data on the RYR1 genetic variability in pig and only the missense mutation Arg615Cys, associated with the malignant hyperthermia, porcine stress syndrome and meat quality, has been studied in several commercial and local breeds. Aim. To genotype the rs344435545 (C1972T, Arg615Cys), rs196953058 (T8434C, Phe2769Leu) and rs323041392 (G12484A, Asp4119Asn) in the Latvian local pig breed Latvian White and to evaluate the ev…

0301 basic medicineGeneticspigGenetic diversityAnimal breedingbiologyQH301-705.5genetic diversityQH426-470biology.organism_classificationGeneral Biochemistry Genetics and Molecular BiologyBreedpolymorphism03 medical and health sciences030104 developmental biologyGenetic variationGenotypeRYR1GeneticsGenomics Transcriptomics and ProteomicsRestriction fragment length polymorphismAlleleBiology (General)Latvian White pigBiopolymers and Cell
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The dark side of centromeres: types, causes and consequences of structural abnormalities implicating centromeric DNA

2018

Centromeres are the chromosomal domains required to ensure faithful transmission of the genome during cell division. They have a central role in preventing aneuploidy, by orchestrating the assembly of several components required for chromosome separation. However, centromeres also adopt a complex structure that makes them susceptible to being sites of chromosome rearrangements. Therefore, preservation of centromere integrity is a difficult, but important task for the cell. In this review, we discuss how centromeres could potentially be a source of genome instability and how centromere aberrations and rearrangements are linked with human diseases such as cancer.

0301 basic medicineGenome instabilityCell division[SDV]Life Sciences [q-bio]ScienceCentromereGeneral Physics and AstronomyAneuploidy[SDV.BC]Life Sciences [q-bio]/Cellular BiologyReview ArticleBiologyChromosomeModels BiologicalGenomeChromosomesGenomic InstabilityGeneral Biochemistry Genetics and Molecular Biology03 medical and health scienceschemistry.chemical_compoundCentromeremedicineHumansDiseaselcsh:ScienceChromosome separationComputingMilieux_MISCELLANEOUSGeneticsMultidisciplinaryQChromosomeDNAGeneral Chemistrymedicine.diseaseSettore BIO/18 - Genetica030104 developmental biologychemistrylcsh:QDNANature Communications
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DNA Damage Signaling Instructs Polyploid Macrophage Fate in Granulomas.

2018

Granulomas are immune cell aggregates formed in response to persistent inflammatory stimuli. Granuloma macrophage subsets are diverse and carry varying copy numbers of their genomic information. The molecular programs that control the differentiation of such macrophage populations in response to a chronic stimulus, though critical for disease outcome, have not been defined. Here, we delineate a macrophage differentiation pathway by which a persistent Toll-like receptor (TLR) 2 signal instructs polyploid macrophage fate by inducing replication stress and activating the DNA damage response. Polyploid granuloma-resident macrophages formed via modified cell divisions and mitotic defects and not…

0301 basic medicineGenome instabilityDNA damageLipoproteinsCellMitosisInflammationAtaxia Telangiectasia Mutated ProteinsBiologymedicine.disease_causeGeneral Biochemistry Genetics and Molecular BiologyProto-Oncogene Proteins c-myc03 medical and health sciencesMicemedicineAnimalsHumansMacrophage Differentiation PathwayMitosisCell ProliferationInflammationGranulomaMacrophagesCell DifferentiationMycobacterium tuberculosisToll-Like Receptor 2Cell biologyMice Inbred C57BLTLR2030104 developmental biologymedicine.anatomical_structureImmunologymedicine.symptomCarcinogenesisDNA DamageCell
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Inhibition of DNA damage response at telomeres improves the detrimental phenotypes of Hutchinson–Gilford Progeria Syndrome

2019

Hutchinson–Gilford progeria syndrome (HGPS) is a genetic disorder characterized by premature aging features. Cells from HGPS patients express progerin, a truncated form of Lamin A, which perturbs cellular homeostasis leading to nuclear shape alterations, genome instability, heterochromatin loss, telomere dysfunction and premature entry into cellular senescence. Recently, we reported that telomere dysfunction induces the transcription of telomeric non-coding RNAs (tncRNAs) which control the DNA damage response (DDR) at dysfunctional telomeres. Here we show that progerin-induced telomere dysfunction induces the transcription of tncRNAs. Their functional inhibition by sequence-specific telomer…

0301 basic medicineGenome instabilityRNA UntranslatedDNA RepairGeneral Physics and AstronomyCellular homeostasisAntisense oligonucleotide therapyMice0302 clinical medicineProgeriaHomeostasislcsh:ScienceCellular SenescenceSkinProgeriaMultidisciplinaryintegumentary systemQTelomereProgerinLamin Type A3. Good healthCell biologyTelomeresPhenotypePremature agingcongenital hereditary and neonatal diseases and abnormalitiesDNA repairScienceDouble-strand DNA breaksBiologySettore MED/08 - Anatomia PatologicaGeneral Biochemistry Genetics and Molecular BiologyArticleCell Line03 medical and health sciencesmedicineDNA damage Hutchinson-Gilford Progeria SyndromeAnimalsCell Proliferationnutritional and metabolic diseasesGeneral ChemistryOligonucleotides Antisensemedicine.diseaseTelomereDisease Models Animal030104 developmental biologyMutationlcsh:Q030217 neurology & neurosurgeryLaminDNA DamageNature Communications
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FANCD2 modulates the mitochondrial stress response to prevent common fragile site instability

2021

Common fragile sites (CFSs) are genomic regions frequently involved in cancer-associated rearrangements. Most CFSs lie within large genes, and their instability involves transcription- and replication-dependent mechanisms. Here, we uncover a role for the mitochondrial stress response pathway in the regulation of CFS stability in human cells. We show that FANCD2, a master regulator of CFS stability, dampens the activation of the mitochondrial stress response and prevents mitochondrial dysfunction. Genetic or pharmacological activation of mitochondrial stress signaling induces CFS gene expression and concomitant relocalization to CFSs of FANCD2. FANCD2 attenuates CFS gene transcription and pr…

0301 basic medicineGenome instabilitymusculoskeletal diseasesTranscription GeneticQH301-705.5RegulatorMedicine (miscellaneous)MitochondrionBiology[SDV.BBM.BM] Life Sciences [q-bio]/Biochemistry Molecular Biology/Molecular biologyGeneral Biochemistry Genetics and Molecular BiologyOxidative PhosphorylationArticle03 medical and health sciences0302 clinical medicineTranscription (biology)Stress Physiologicalhemic and lymphatic diseasesGene expressionFANCD2HumansBiology (General)GeneUbiquitinsChromosomal fragile siteChromosome Fragile SitesChromosome FragilityFanconi Anemia Complementation Group D2 ProteinDNA damage and repair[SDV.BBM.BM]Life Sciences [q-bio]/Biochemistry Molecular Biology/Molecular biologyHCT116 CellsCell biologyMitochondriaSettore BIO/18 - Genetica030104 developmental biologyGene Expression Regulation030220 oncology & carcinogenesisUnfolded Protein ResponseGeneral Agricultural and Biological SciencesDNA Damage
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Evolutionary impact of copy number variation rates.

2017

[Objective]: Copy number variation is now recognized as one of the major sources of genetic variation among individuals in natural populations of any species. However, the relevance of these unexpected observations goes beyond diagnosing high diversity. [Results]: Here, it is argued that the molecular rates of copy number variation, mainly the deletion rate upon variation, determine the evolutionary road of the genome regarding size. Genetic drift will govern this process only if the efective population size is lower than the inverse of the deletion rate. Otherwise, natural selection will do.

0301 basic medicineGenome sizeDNA Copy Number VariationsGene duplicationPopulation geneticsPopulation geneticslcsh:MedicineBiologyGeneral Biochemistry Genetics and Molecular Biology03 medical and health sciencesEffective population sizeGenetic driftGenetic variationAnimalsHumansCopy-number variationlcsh:Science (General)Genome sizelcsh:QH301-705.5GeneticsNatural selectionlcsh:RGenetic DriftBirth–death processGeneral MedicineBiological EvolutionResearch Note030104 developmental biologyGenetics Populationlcsh:Biology (General)Evolutionary biologyNeutral theory of molecular evolutionNeutral evolutionlcsh:Q1-390BMC research notes
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Predictive ability of the Mini Nutritional Assessment Short Form (MNA-SF) in a free-living elderly population: a cross-sectional study

2017

Background Various scales have been used to perform a quick and first level nutritional assessment, and the MNA is one of the most used and recommended by experts in the elderly in all areas. This scale has a short form, the MNA-SF, revised and validated in 2009, which has two versions: the BMI-MNA-SF contains the first six items of the full scale including Body Mass Index while the CC-MNA-SF includes Calf Circumference instead of BMI. Objective To evaluate the predictive ability for nutritional status of the two versions of the MNA-SF against the MNA in free-living elderly in the province of Valencia. Methods Cross-sectional study of 660 free-living elderly in the province of Valencia sel…

0301 basic medicineGerontologyFree-living elderlyCross-sectional studyESTADISTICA E INVESTIGACION OPERATIVAPopulationlcsh:MedicineNursingGeneral Biochemistry Genetics and Molecular Biology03 medical and health sciences0302 clinical medicineMedicine030212 general & internal medicineeducationNutritionRank correlationeducation.field_of_study030109 nutrition & dieteticsReceiver operating characteristicbusiness.industryGeneral Neurosciencelcsh:RGeneral MedicineGold standard (test)Nutritional assessmentMNA-SFmedicine.diseaseMalnutritionGeriatricsEnfermeríaGeneral Agricultural and Biological SciencesbusinessBody mass indexKappaDemographyPeerJ
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Special Issue “Mental Health Issues and Quality of Life in Older Individuals”

2021

There are numerous biological, psychological, and social factors that have a more or less prominent impact on the mental health of older adults [...]

0301 basic medicineGerontologyPaleontologyMental healthGeneral Biochemistry Genetics and Molecular Biology03 medical and health sciencesEditorialn/a030104 developmental biology0302 clinical medicineQuality of life (healthcare)Space and Planetary Sciencelcsh:Qlcsh:SciencePsychology030217 neurology & neurosurgeryEcology Evolution Behavior and SystematicsLife
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Oligodendrocytes Support Neuronal Glutamatergic Transmission via Expression of Glutamine Synthetase.

2019

Summary: Glutamate has been implicated in a wide range of brain pathologies and is thought to be metabolized via the astrocyte-specific enzyme glutamine synthetase (GS). We show here that oligodendrocytes, the myelinating glia of the central nervous system, also express high levels of GS in caudal regions like the midbrain and the spinal cord. Selective removal of oligodendrocyte GS in mice led to reduced brain glutamate and glutamine levels and impaired glutamatergic synaptic transmission without disrupting myelination. Furthermore, animals lacking oligodendrocyte GS displayed deficits in cocaine-induced locomotor sensitization, a behavior that is dependent on glutamatergic signaling in th…

0301 basic medicineGlutamineCentral nervous systemNeurotransmissionBiologyGeneral Biochemistry Genetics and Molecular BiologyArticleMidbrain03 medical and health sciencesGlutamatergic0302 clinical medicineGlutamate-Ammonia LigaseGlutamine synthetasemedicineAnimalslcsh:QH301-705.5Glutamate receptorBrainOligodendrocyteCell biologyGlutamineOligodendroglia030104 developmental biologymedicine.anatomical_structurenervous systemlcsh:Biology (General)030217 neurology & neurosurgerySignal TransductionCell reports
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